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Prevalence
Between 3-5% of population is affected.
Ratio of male to female is 3:1 worldwide, and up to 12:1 in Australia (Wallace,
1999).
Aetiology
Neurological studies have found abnormal prefrontal activity in ADHD (Monastra et al, 1999).
Research suggests a genetic link. Up to 90% of ADHD children have a family history of ADHD or related disorder.
Neurochemical and neurobiological abnormalities are the most accepted causes (Barkley, 1997).In some cases prenatal
exposure to lead or other toxins is implicated.
Food allergies and sensitivity to additives or colouring is not causally related, however in some cases, a modified
diet can alleviate symptoms of hyperactivity.
Early childhood environment, including parent-child relationships are not causally related, but may exasperate symptoms
(especially the development of disruptive or oppositional defiance disorder).
Diagnostic Criteria
Poor sustained attention.
Children with ADHD are typically very easily distracted and
they have difficulty ‘staying on task’. In school and throughout their adult lives, these individuals fail to
finish tasks, fail to follow through with instructions, fail to pay attention when being spoken to, and remain disorganised
in daily living. These children ‘forget’ important items necessary for school (E.g. pens, erasers, books, warm
jumper, or bus pass).
Lack of impulse control.
Children with ADHD do not necessarily fail to think before
they act, but they do lack the ability to inhibit actions. In school, impulsive behaviours may be evident; blurting out answers,
repeatedly breaking school rules, continual talking in class and risk-taking behaviours. Young children may impulsively run
out in front of traffic to get a ball, whilst older ADHD children are easily persuaded to try dangerous hobbies and habits
(E.g. jumping off buildings, or illicit drugs use). However, it is emphasised that these impulsive actions are not malicious or pre-planned (Wallace, 1999).
Hyperactivity.
Children with ADHD often fidget or wriggle in their seat. Difficulty remaining still for any length of
time persists into adulthood, but as these children mature, gross motor movements may decline. Not all ADHD children are hyperactive,
some may even present hypo-activity (lethargy or under activity).
Social skills deficits.
Although not a diagnostic criteria, almost all of these children will have poor social skills. They may have younger
or older friends, but difficulty ‘fitting’ in with their same-age peers. Due to their ‘clowning’ behaviour
in class, younger children with ADHD may have a larger group of ‘friends’,
but become increasingly isolated as their friends ‘grow up’ and seemingly tire of the ADHD adolescents’
antics.
Comorbidity
(other disorders that may co-occur with ADHD)
Learning Disabilities.
About 20 % to 30 % of children with ADHD will also suffer from
a specific learning disability (LD). They may have a phonological processing deficit, central auditory processing deficit, reading
disabilities, dyslexia, problems with writing, and arithmetic disorders
Oppositional Defiant
Disorder.
Between
30% to 50% of children with ADHD have an oppositional defiant disorder (ODD). These children are defiant, stubborn, non-compliant,
have outbursts of temper, argue with adults and refuse to obey rules.
Conduct Disorder.
About 20 to 40 percent
of ADHD children may develop conduct disorder (CD), which ia a pattern of antisocial behaviour. These children bully others,
lie and steal, are aggressive toward people and animals, destroy property, engage in vandalism. As teenagers, they are
at greater risk of substance abuse and juvenile delinquency.
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